Tissue Scurvy and the Triad

Michael D. Innis

doi:doi:10.11648/j.cmr.20140304.13

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Tissue Scurvy and the Triad

Michael D. Innis

Published in Clinical Medicine Research (Volume 3, Issue 4)

Received: 4 July 2014 Accepted: 16 July 2014 Published: 30 July 2014

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Abstract

The Triad, characterized by bilateral retinal haemorrhages, diffuse cerebral haemorrhage and hypoxic ischaemic encephalopathy is conventionally thought to result from trauma caused by violently shaking a child by a parent or carer. The shaking is said to cause a to and fro movement of the brain in the skull leading to injury to the neurons, disruption of the cerebral veins and trauma to the retinal vessels. Vigorous shaking of a child has never been observed and an alternative explanation for these changes was sought. Here it is shown that Tissue Scurvy, an autoimmune disorder which has specific and consistent biochemical features of Hyperglycaemia and Liver Dysfunction as shown by abnormal Liver Function Tests, is the cause of the lesions. A child diagnosed as being the victim of the Shaken Baby Syndrome was tested for evidence of Tissue Scurvy and both Hyperglycaemia and Abnormal Liver Function were demonstrated. It is concluded that these results provide conclusive evidence that the Shaken Baby Syndrome is a form of Tissue Scurvy in which encephalopathy is caused by damage to neurons in the cervical spinal column causing hypoxia, which leads to ischaemic encephalopathy and the other features of the triad.

Published in	Clinical Medicine Research (Volume 3, Issue 4)
DOI	10.11648/j.cmr.20140304.13
Page(s)	94-95
Creative Commons	This is an Open Access article, distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution and reproduction in any medium or format, provided the original work is properly cited.
Copyright	Copyright © The Author(s), 2024. Published by Science Publishing Group

Keywords

Triad, Non-Accidental Injury, Shaken Baby Syndrome, Haemorrhage

References

[1]	Kalokerinos A. “Every Second Child” Thomas Nelson (Australia) Limited 1974
[2]	Innis MD. “Vitamin K Deficiency Disease”. Jour of Orthomol Med 2008;vol 23; 15-20
[3]	Clemetson CAB. “Vaccinations, Inoculations and Ascorbic Acid”. Jour Ortho Mol Med 1999;14:137 –142
[4]	Innis M D. Autoimmune Tissue Scurvy Misdiagnosed as Child Abuse. Clinical Medicine Research. Vol. X, No. X, 2013.
[5]	Cunningham JJ. “The Glucose/Insulin System and Vitamin C: Implications in Insulin-dependent Diabetes Mellitus.” J Am Coll Nutr; 1998:vol 17 p105-108
[6]	Wallin R Vitamin K-dependent carboxylation and vitamin K metabolism in liver. Effects of warfarin. J Clin Invest › v.76(5); Nov 1985.
[7]	Michael D Innis Autoimmunity and non-Accidental Injury in Children Clinical Medicine Research. MM-DD-2013, pp. x-x. doi:10.11648/j.xxx.

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Michael D. Innis. (2014). Tissue Scurvy and the Triad. Clinical Medicine Research, 3(4), 94-95. https://doi.org/10.11648/j.cmr.20140304.13

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ACS Style

Michael D. Innis. Tissue Scurvy and the Triad. Clin. Med. Res. 2014, 3(4), 94-95. doi: 10.11648/j.cmr.20140304.13

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AMA Style

Michael D. Innis. Tissue Scurvy and the Triad. Clin Med Res. 2014;3(4):94-95. doi: 10.11648/j.cmr.20140304.13

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@article{10.11648/j.cmr.20140304.13,
  author = {Michael D. Innis},
  title = {Tissue Scurvy and the Triad},
  journal = {Clinical Medicine Research},
  volume = {3},
  number = {4},
  pages = {94-95},
  doi = {10.11648/j.cmr.20140304.13},
  url = {https://doi.org/10.11648/j.cmr.20140304.13},
  eprint = {https://article.sciencepublishinggroup.com/pdf/10.11648.j.cmr.20140304.13},
  abstract = {The Triad, characterized by bilateral retinal haemorrhages, diffuse cerebral haemorrhage and hypoxic ischaemic encephalopathy is conventionally thought to result from trauma caused by violently shaking a child by a parent or carer. The shaking is said to cause a to and fro movement of the brain in the skull leading to injury to the neurons, disruption of the cerebral veins and trauma to the retinal vessels. Vigorous shaking of a child has never been observed and an alternative explanation for these changes was sought. Here it is shown that Tissue Scurvy, an autoimmune disorder which has specific and consistent biochemical features of Hyperglycaemia and Liver Dysfunction as shown by abnormal Liver Function Tests, is the cause of the lesions. A child diagnosed as being the victim of the Shaken Baby Syndrome was tested for evidence of Tissue Scurvy and both Hyperglycaemia and Abnormal Liver Function were demonstrated. It is concluded that these results provide conclusive evidence that the Shaken Baby Syndrome is a form of Tissue Scurvy in which encephalopathy is caused by damage to neurons in the cervical spinal column causing hypoxia, which leads to ischaemic encephalopathy and the other features of the triad.},
 year = {2014}
}

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TY  - JOUR
T1  - Tissue Scurvy and the Triad
AU  - Michael D. Innis
Y1  - 2014/07/30
PY  - 2014
N1  - https://doi.org/10.11648/j.cmr.20140304.13
DO  - 10.11648/j.cmr.20140304.13
T2  - Clinical Medicine Research
JF  - Clinical Medicine Research
JO  - Clinical Medicine Research
SP  - 94
EP  - 95
PB  - Science Publishing Group
SN  - 2326-9057
UR  - https://doi.org/10.11648/j.cmr.20140304.13
AB  - The Triad, characterized by bilateral retinal haemorrhages, diffuse cerebral haemorrhage and hypoxic ischaemic encephalopathy is conventionally thought to result from trauma caused by violently shaking a child by a parent or carer. The shaking is said to cause a to and fro movement of the brain in the skull leading to injury to the neurons, disruption of the cerebral veins and trauma to the retinal vessels. Vigorous shaking of a child has never been observed and an alternative explanation for these changes was sought. Here it is shown that Tissue Scurvy, an autoimmune disorder which has specific and consistent biochemical features of Hyperglycaemia and Liver Dysfunction as shown by abnormal Liver Function Tests, is the cause of the lesions. A child diagnosed as being the victim of the Shaken Baby Syndrome was tested for evidence of Tissue Scurvy and both Hyperglycaemia and Abnormal Liver Function were demonstrated. It is concluded that these results provide conclusive evidence that the Shaken Baby Syndrome is a form of Tissue Scurvy in which encephalopathy is caused by damage to neurons in the cervical spinal column causing hypoxia, which leads to ischaemic encephalopathy and the other features of the triad.
VL  - 3
IS  - 4
ER  -

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Author Information

Michael D. Innis

Retired Haematologist, Princess Alexandra Hospital, Brisbane, Australia

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Plain Text BibTeX RIS

APA Style

Michael D. Innis. (2014). Tissue Scurvy and the Triad. Clinical Medicine Research, 3(4), 94-95. https://doi.org/10.11648/j.cmr.20140304.13

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ACS Style

Michael D. Innis. Tissue Scurvy and the Triad. Clin. Med. Res. 2014, 3(4), 94-95. doi: 10.11648/j.cmr.20140304.13

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AMA Style

Michael D. Innis. Tissue Scurvy and the Triad. Clin Med Res. 2014;3(4):94-95. doi: 10.11648/j.cmr.20140304.13

Copy | Download

@article{10.11648/j.cmr.20140304.13,
  author = {Michael D. Innis},
  title = {Tissue Scurvy and the Triad},
  journal = {Clinical Medicine Research},
  volume = {3},
  number = {4},
  pages = {94-95},
  doi = {10.11648/j.cmr.20140304.13},
  url = {https://doi.org/10.11648/j.cmr.20140304.13},
  eprint = {https://article.sciencepublishinggroup.com/pdf/10.11648.j.cmr.20140304.13},
  abstract = {The Triad, characterized by bilateral retinal haemorrhages, diffuse cerebral haemorrhage and hypoxic ischaemic encephalopathy is conventionally thought to result from trauma caused by violently shaking a child by a parent or carer. The shaking is said to cause a to and fro movement of the brain in the skull leading to injury to the neurons, disruption of the cerebral veins and trauma to the retinal vessels. Vigorous shaking of a child has never been observed and an alternative explanation for these changes was sought. Here it is shown that Tissue Scurvy, an autoimmune disorder which has specific and consistent biochemical features of Hyperglycaemia and Liver Dysfunction as shown by abnormal Liver Function Tests, is the cause of the lesions. A child diagnosed as being the victim of the Shaken Baby Syndrome was tested for evidence of Tissue Scurvy and both Hyperglycaemia and Abnormal Liver Function were demonstrated. It is concluded that these results provide conclusive evidence that the Shaken Baby Syndrome is a form of Tissue Scurvy in which encephalopathy is caused by damage to neurons in the cervical spinal column causing hypoxia, which leads to ischaemic encephalopathy and the other features of the triad.},
 year = {2014}
}

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TY  - JOUR
T1  - Tissue Scurvy and the Triad
AU  - Michael D. Innis
Y1  - 2014/07/30
PY  - 2014
N1  - https://doi.org/10.11648/j.cmr.20140304.13
DO  - 10.11648/j.cmr.20140304.13
T2  - Clinical Medicine Research
JF  - Clinical Medicine Research
JO  - Clinical Medicine Research
SP  - 94
EP  - 95
PB  - Science Publishing Group
SN  - 2326-9057
UR  - https://doi.org/10.11648/j.cmr.20140304.13
AB  - The Triad, characterized by bilateral retinal haemorrhages, diffuse cerebral haemorrhage and hypoxic ischaemic encephalopathy is conventionally thought to result from trauma caused by violently shaking a child by a parent or carer. The shaking is said to cause a to and fro movement of the brain in the skull leading to injury to the neurons, disruption of the cerebral veins and trauma to the retinal vessels. Vigorous shaking of a child has never been observed and an alternative explanation for these changes was sought. Here it is shown that Tissue Scurvy, an autoimmune disorder which has specific and consistent biochemical features of Hyperglycaemia and Liver Dysfunction as shown by abnormal Liver Function Tests, is the cause of the lesions. A child diagnosed as being the victim of the Shaken Baby Syndrome was tested for evidence of Tissue Scurvy and both Hyperglycaemia and Abnormal Liver Function were demonstrated. It is concluded that these results provide conclusive evidence that the Shaken Baby Syndrome is a form of Tissue Scurvy in which encephalopathy is caused by damage to neurons in the cervical spinal column causing hypoxia, which leads to ischaemic encephalopathy and the other features of the triad.
VL  - 3
IS  - 4
ER  -

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