Alzheimer’s Disease
Submission Deadline: Nov. 25, 2019

This special issue currently is open for paper submission and guest editor application.

Join as Guest Editor Submit to Special Issue
Lead Guest Editor
Mehmet Bostancıklıoğlu
Gaziantep University, Gaziantep, Turkey
Guest Editors
  • Ebru Temiz
    Harran University, Şanlıurfa, Turkey
  • İbrahim Bozgeyik
    Adiyaman University, Adıyaman, Turkey
  • Esra Bozgeyik
    Tekirdağ Namık Kemal University, Tekirdağ, Turkey
  • Mustafa Ulaşlı
    Gaziantep University, Gaziantep, Turkey
  • Serdar Öztuzcu
    Gaziantep University, Gaziantep, Turkey
  • Beyhan Cengiz
    Gaziantep University, Gaziantep, Turkey
  • Tuncer Demir
    Gaziantep University, Gaziantep, Turkey
  • Yusuf Ziya İğci
    Gaziantep University, Gaziantep, Turkey
  • Mehri İğci
    Gaziantep University, Gaziantep, Turkey
  • Meriç Temiz
    Gaziantep University, Gaziantep, Turkey
Guidelines for Submission
Manuscripts can be submitted until the expiry of the deadline. Submissions must be previously unpublished and may not be under consideration elsewhere.
Papers should be formatted according to the guidelines for authors (see: By submitting your manuscripts to the special issue, you are acknowledging that you accept the rules established for publication of manuscripts, including agreement to pay the Article Processing Charges for the manuscripts. Manuscripts should be submitted electronically through the online manuscript submission system at All papers will be peer-reviewed. Accepted papers will be published continuously in the journal and will be listed together on the special issue website.
Published Papers
The special issue currently is open for paper submission. Potential authors are humbly requested to submit an electronic copy of their complete manuscript by clicking here.

Special Issue Flyer (PDF)

Please download to know all details of the
Special Issue

Alzheimer’s disease is characterized by severe deficits in memory, cognition and motor functions, and the most prominent histopathological clues are the aggregation of amyloid-beta peptides and hyperphosphorylated tau tangles. In addition, other hypothesis explaining the pathogenesis of AD highlight neuroinflammation, an impairment in the calcium balance and energy metabolism, and vascular degeneration, but therapeutic strategies based on these current theories and ideas have not yielded any benefits during the last decades. This special issue aims to improve the dissemination of advanced research in the area of Alzheimer’s disease. Original research papers are solicited in any aspect of Alzheimer’s disease.

Aims and Scope:

  1. Interactions between gut microbiota and Alzheimer’s disease
  2. Explaining the pathogenesis of Alzheimer’s disease
  3. Memory retrieval effects of engram cells in Alzheimer’s disease
  4. Neuroinflammation
  5. Protein clearance mechanisms
  6. Antibody usage to prevent amyloid beta plaque accumulation
  7. Amyloid Beta hypothesis
  8. Acetylcholine hypothesis
  9. Neurotransmitter systems affect memory and other cognitive functions
  10. New animal models of Alzheimer’s disease
  11. Animal models
  12. The effect of selective serotonin receptor inhibitor on memory
  13. Learning and memory formation
  14. Memory retrieval
  15. Calcium and energy metabolism
  16. Vascular degeneration
  17. New treatment approaches
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