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Alzheimer’s disease is characterized by severe deficits in memory, cognition and motor functions, and the most prominent histopathological clues are the aggregation of amyloid-beta peptides and hyperphosphorylated tau tangles. In addition, other hypothesis explaining the pathogenesis of AD highlight neuroinflammation, an impairment in the calcium balance and energy metabolism, and vascular degeneration, but therapeutic strategies based on these current theories and ideas have not yielded any benefits during the last decades. This special issue aims to improve the dissemination of advanced research in the area of Alzheimer’s disease. Original research papers are solicited in any aspect of Alzheimer’s disease.
Aims and Scope:
Interactions between gut microbiota and Alzheimer’s disease
Explaining the pathogenesis of Alzheimer’s disease
Memory retrieval effects of engram cells in Alzheimer’s disease
Protein clearance mechanisms
Antibody usage to prevent amyloid beta plaque accumulation
Amyloid Beta hypothesis
Neurotransmitter systems affect memory and other cognitive functions
New animal models of Alzheimer’s disease
The effect of selective serotonin receptor inhibitor on memory