Inflammatory Bowel Disease
Submission Deadline: May 25, 2020

This special issue currently is open for paper submission and guest editor application.

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Lead Guest Editor
Nirmal Verma
Department of Pharmacology and Nutritional Sciences, University of Kentucky, Lexington, Kentucky, USA
Guest Editors
  • Ravi Verma
    Pohang University of Science and Technology
    Gyeongbuk, South Korea
  • Anil Verma
    Indian Council of Medical Research
    New Delhi, India
  • Reena Kumari
    University of Kentucky
    Kentucky, USA
  • Raju Ranjha
    National Institute of Malaria Research
    Rourkela, India
  • Naresh Kumar Meena
    NIH
    Bethesda, USA
  • Jaishree Paul
    Jawaharlal Nehru University
    New Delhi, India
Guidelines for Submission
Manuscripts can be submitted until the expiry of the deadline. Submissions must be previously unpublished and may not be under consideration elsewhere.
Papers should be formatted according to the guidelines for authors (see: http://www.sciencepublishinggroup.com/journal/guideforauthors?journalid=655). By submitting your manuscripts to the special issue, you are acknowledging that you accept the rules established for publication of manuscripts, including agreement to pay the Article Processing Charges for the manuscripts. Manuscripts should be submitted electronically through the online manuscript submission system at http://www.sciencepublishinggroup.com/login. All papers will be peer-reviewed. Accepted papers will be published continuously in the journal and will be listed together on the special issue website.
Published Papers
The special issue currently is open for paper submission. Potential authors are humbly requested to submit an electronic copy of their complete manuscript by clicking here.

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Special Issue

Introduction
Ulcerative colitis (UC) and Crohn’s disease (CD) are chronic inflammatory disorders of the intestinal tract. They are summarized as inflammatory bowel disease (IBD). In UC, the inflammation occurs in the mucosa and mucosal ulceration can develop. UC is mainly localized in the rectum and spreads to proximal parts of the intestine to a different extent. In CD, any part of the gastrointestinal tract can be affected, however the main site of inflammation is the terminal ileum and inflammation can occur segmental and discontinuously. In contrast to UC, not only the mucosa is affected but also all layers of the intestinal wall and granuloma are formed in CD. The pathogenesis of both diseases is not yet fully elucidated. Different factors such as genetics, immune dysregulation, and the microbial flora in the intestine and barrier dysfunction of intestinal epithelial cells may lead to the pathology of IBD. The pathogenesis of IBD is poorly understood because of the variability in clinical manifestations and complexity of the mechanisms of chronic inflammation.
There is increasing evidence that IBD tissue injury involves many cell types present in the bowel wall. The epithelial cell has been traditionally considered an important component of IBD pathogenesis, especially in UC. Due to the variability, complexity, and chronicity of gut inflammation in IBD, intestinal epithelial cells (IEC) constantly adapt to the multiple events occurring in the mucosal microenvironment, a phenomenon that has been long recognized. This adaptation might cause IEC to undergo changes in growth and differentiation, metabolism, secretary pattern, immune function, and antigen expression.
Taking into account the practical importance of searching for new effective approaches for Inflammatory Bowel Diseases treatment and prevention, we dedicate a special issue of the journal to this problem.

Aims and Scope:

  1. Ulcerative colitis
  2. Crohn’s disease
  3. Gut Immunology
  4. Gut genetic factors
  5. Gut Microbiota
  6. Gut micro RNA
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